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A Comprehensive Anemia Formulation

Anem-X is a full-service anemia formulation providing all vitamins and minerals whose deficiency may cause anemia. These nutrients are provided in the most bioavailable form to create the most immediate impact to the underlining anemia.


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Product Description

The Anem-X Difference - Managing All Forms of Anemia

• Contains the highly absorbable ferrous form of iron. All dietary iron has to be reduced to the ferrous form for absorption. Ferrous iron is absorbed three times more readily than the ferric form providing faster acting anemia support.

• Contains the active form of folate ((L-5-MTHF (L-5-methyltetrahydrofolate) instead of folic acid)) to ensure optimal bioavailability. Up to 20% of Americans have a genetic imbalance preventing the conversion of folic acid. The folate in Anem-X does not need to be converted.

• Contains the active form of B12 (methylcobalamin instead of cyanocobalamin) which can prevent or reverse B12 deficiency related anemias.

• Contains the active form of B6 (P-5-P, Pyridoxine-5-Phosphate) which is used as a coenzyme in the production of both red and white blood cells. Conversion of inactive Pyridoxine HCL to P-5-P can be inhibited by faulty immune system, kidney and liver problems, cancer and numerous forms of medications.

• Contains niacin which plays a positive role in the management of anemia, promotes healthy circulation and assists in the creation of energy needed to form red blood cells.

• Contains copper which is required for proper iron absorption and utilization. It enhances the development of red and white blood cells. Copper deficiency can lead to low levels of iron. Government statistics indicate that, on average, Americans consume only 50% to 60% of the RDA for copper.* Utilize a well-rounded, complete anemia formulation designed for fasting-acting results from all forms of nutrient-related anemias.

*These statements have not been evaluated by the Food and Drug Administration. This product is not intended to diagnose, treat, cure or prevent any disease.

Supplemental Facts

Supplemental Facts


Serving Size: 1 Tablet


Servings per Container: 120





% Daily Value





Niacin (as niacinamide)




Vitamin B6 (as pyridoxal 5-phosphate)




Folate (as 5-MTHF)




Vitamin B12 (as methylcobalamin)




Iron (as ferrous glycinate)




Copper ( as copper gluconate)




Other Ingredients:  Dicalcium phosphate, microcrystalline cellulose, stearic acid, film coat (hypromellose, polyethylene glycol, hydroxypropyl cellulose), croscarmellose sodium, vegetable stearate and silica



† Daily Value not established by the FDA






Suggested Use

Suggested Use

Suggested Use:

As a dietary supplement, take one (1) tablet daily with food or as directed by your health care professional.


As with any dietary supplement, consult your healthcare practitioner before using this product, especially if you are pregnant, nursing, anticipate surgery, take any medication or are otherwise under medical supervision. Accidental overdose of iron-containing products is a leading cause of fatal poisoning in children under 6. Keep this product out of reach of children. In case of accidental overdose, call a doctor or poison control center immediately. 

Formulated to Be Free of Allergens Derived From:

Wheat, gluten, soy, dairy, eggs, fish, crustacean shellfish, tree nuts, peanuts, artificial preservatives, sweeteners, color and flavors. 

* These statements have not been evaluated by the Food and Drug Administration. This product is not intended to diagnose, treat, cure or prevent any disease.




Sources and Research Abstracts Supporting Anem-X

1. Little DR. Ambulatory management of common forms of anemia. Am Fam Physician 1999;59:1598-604 [review]

2. Hirschmann JV, Raugi GJ. Adult scurvy. J Am Acad Dermatol 1999;41:895-906 [review].

3. Orehek AJ, Kollas CD. Refractory postpartum anemia due to vitamin B6 defi ciency. Ann Intern Med1997;126(10):834-5 [letter].

4. Iwama H, Iwase O, Hayashi S, et al. Macrocytic anemia with anisocytosis due to alcohol abuse and vitamin B6 defi ciency. Rinsho Ketsueki 1998;39:1127-30 [in Japanese].

5. Summerfi eld AL, Steinberg FU, Gonzalez JG. Morphologic fi ndings in bone marrow precursor cells in zinc-induced copper defi ciency anemia. Am J Clin Pathol 1992;97:665-8.

6. Freycon F, Pouyau G. Rare nutritional defi ciency anemia: defi ciency of copper and vitamin E. Sem Hop1983;59:488- 93 [review] [in French].

7. Samuels, AJ. Studies in patients with functional menorrhagia: the antimenorrhagic effect of the adequate replication of iron stores. Isr J Med Sci 1965;1:851-3.

8. Taymor ML, Sturgis SH, Yahia C. The etiological role of chronic iron defi ciency in production of menorrhagia.JAMA 1964;187:323-7.

9. Vaucher P, Druais PL, Waldvogel S, Favrat B. Effect of iron supplementation on fatigue in nonanemic menstruating women with low ferritin: a randomized controlled trial. CMAJ 2012;184:1247–54.

10. Romslo I, Haram K, Sagen N, Augensen K. Iron requirement in normal pregnancy as assessed by serum ferritin, serum transferrin saturation and erythrocyte protoporphyrin determinations. Br J Obstet Gynaecol1983;90:101-7.

11. Palacios Santiago, “Ferrous versus Ferric Oral Iron Formulations for the Treatment of Iron Defi ciency: A Clinical Overview,” The Scientifi c World Journal, vol. 2012, Article ID 846824, 5 pages, 2012. doi:10.1100/2012/846824

12. Wilcken B, Bamforth F, Li Z, et al. Geographical and ethnic variation of the 677C>T allele of 5,10 methylenetetrahydrofolate reductase (MTHFR): fi ndings from over 7000 newborns from 16 areas world wide. J Med Genet. 2003;40(8):619-625.


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